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cailynn johnson
Oligosaccharide synthesis enables the production of structurally defined sugar chains crucial for studying cell signaling, immune responses, and disease mechanisms. Using chemical or enzymatic methods, researchers can create pure oligosaccharides for vaccine development, glycan profiling, and therapeutic research. These synthetic glycans help overcome challenges in isolating natural forms and support innovations in glycobiology and glycomedicine.
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Polysaccharide synthesis enables the creation of complex sugar-based biomaterials with tailored structures and functions. Through chemical strategies like step-wise glycosylation and ring-opening polymerization, scientists can design polysaccharides with specific branching, linkages, and molecular weights. These custom materials are vital for biomedical applications, offering unique viscoelastic and biological properties for drug delivery, tissue engineering, and therapeutic innovation.
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Glycoprotein synthesis is essential for producing homogeneous proteins with defined glycan structures, crucial for studying biological functions and therapeutic development. Using chemical and enzymatic methods, scientists can construct glycoproteins with precise glycosylation patterns. These techniques help explore protein stability, efficacy, and interactions, offering valuable insights into immune responses, cell signaling, and the design of next-generation biologics.
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Parkinson’s disease (PD) in vitro models provide a controlled environment to study dopaminergic neuron degeneration, α-synuclein aggregation, and neuroinflammation. These models utilize iPSC-derived neurons, midbrain organoids, and primary cultures to replicate disease mechanisms and test therapeutic strategies. Techniques such as CRISPR gene editing, high-content screening, and biomarker analysis enable researchers to explore drug efficacy, mitochondrial dysfunction, and immune interactions. Advancing PD in vitro models enhances precision medicine, supporting innovative treatment development.
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Alzheimer’s disease (AD) in vitro models provide a controlled environment to study amyloid-beta (Aβ) plaque formation, tau pathology, and neuroinflammation. These models utilize iPSC-derived neurons, astrocytes, and microglia to replicate disease mechanisms and test therapeutic strategies. Techniques such as 3D organoids, high-content screening, and biomarker analysis enable researchers to explore drug efficacy, synaptic dysfunction, and blood-brain barrier integrity.
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